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Download Cinemascope - Issue 42 - Spring 2010 issue 42 by Mark Peranson (Editor and Publisher) PDF

By Mark Peranson (Editor and Publisher)

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Morris DG et al. Identification of adrenocorticotropin receptor messenger ribonucleic acid in the human pituitary and its loss of expression in pituitary adenomas. J Clin Endocrinol Metab. 2003;88:6080–7. 66. Bamberger CM, Schulte HM, Chrousos GP. Molecular determinants of glucocorticoid receptor function and tissue sensitivity to glucocorticoids. Endocr Rev. 1996;17:245–61. 67. Dahia PL et al. Expression of glucocorticoid receptor gene isoforms in corticotropin-secreting tumors. J Clin Endocrinol Metab.

The secretory granules are usually abundant and vary in electron density, shape, and size, ranging from 150 to 450 nm. The secretory granules tend to accumulate under the cell membrane. 1f). Adenomas of Nelson’s Syndrome This type of adenoma is found in the patients who have undergone bilateral adrenalectomy in order to control refractory glucocorticoid overproduction. It is hypothesized that lack of glucocorticoid negative feedback following bilateral adrenalectomy is associated with the progression of a pre-existing corticotroph adenoma [7].

Silent subtype 1 adenomas are more differentiated and have similar histologic, immunohistochemical, and ultrastructural features to functional corticotroph adenomas. 1). Ultrastructurally, silent corticotroph adenomas have two different subtypes. Silent subtype 1 adenomas resemble those of functional corticotroph adenomas, while silent type 2 adenomas have less differentiated ultrastructural features. The cells of subtype 2 are smaller in size and have a polyhedral shape with centrally placed nuclei.

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