By Intensive Care Society Staff
This factor of the severe care concentration sequence addresses matters in terms of gastro-intestinal platforms suitable to seriously in poor health sufferers at the extensive Care Unit. It comprises chapters in acute pancreatis, diarrohea, intestine finction in the course of enteral feeding, administration of fistulae and haemorrhage and the physiology/pathoplogy of mesenteric ischaemia. additionally, the function of the intestine within the improvement of distant organ failure is mentioned.
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Additional info for Critical Care Focus 9: Gut
Lancet 1999;353:1838–42. Cabie A, Farkas JC, Fitting C, et al. High levels of portal TNF-alpha during abdominal aortic surgery in man. Cytokine 1993;5:448–53. Sanchez-Garcia M, Prieto A, Tejedor A, et al. Characteristics of thoracic duct lymph in multiple organ dysfunction syndrome. Arch Surg 1997;132:13–18. Dubos RJ, Schaedler RW. The effect of the intestinal flora on the growth rate of mice, and their susceptibility to experimental infections. J Exp Med 1960;111: 407–17. THE GUT AS THE MOTOR OF ORGAN FAILURE 18 Neish AS, Gewirtz AT, Zeng H, et al.
Post-operative sepsis was also more common in these patients. Bacterial translocation occurred in 21% of patients but fungal translocation did not occur. It was concluded that proximal gut colonisation was associated with both increased bacterial translocation and septic morbidity. To examine the spectrum of bacteria involved in translocation in surgical patients undergoing laparotomy and to determine the relation between nodal migration of bacteria and the development of post-operative septic complications O’Boyle et al.
Clinical nutrition in pancreatitis. Dig Dis Sci 1997;42:2035–44. 24 Kalfarentzos F, Kehagias J, Mead N, Kokkinis K, Gogos CA. Enteral nutrition is superior to parenteral nutrition in severe acute pancreatitis: results of a randomized prospective trial. Br J Surg 1997;84:1665–9. 25 Windsor AC, Kanwar S, Li AG, et al. Compared with parenteral nutrition, enteral feeding attenuates the acute phase response and improves disease severity in acute pancreatitis. Gut 1998;42:431–5. 26 Pupelis G, Austrums E, Jansone A, Sprucs R, Wehbi H.